A criminally underrated aspect of their music is just how catchy their music is. Everyone loves to talk about their "Big, Scary, Weird" aggression, but their true genius was how effortlessly they would pack every single song to the brim with different and memorable hooks. Whether it be from the texture or the melody itself, they have an impeccable ability to essentially craft memes in music form. I've been a fan since Exmilitary and every time an album dropped, my friends and I would basically just recite our favorite moments during a drinking session and it would devolve into us energetically miming and reciting melodies and lyrics for hours. What a band

ability to essentially craft memes in music form.

Shortly after Munroe's blog post about the script went live, 4chan administrator Christopher Poole adapted the script to moderate the site's experimental /r9k/ board.[23] Twitch offers Robot9000 ("r9k mode") as an optional feature for broadcasters and moderators to use in their channels' chat boxes.[24]

In early 2010, Munroe ran the xkcd Color Name Survey, in which participants were shown a series of RGB colors and asked to enter a suitable name for each specific color. Munroe wanted to identify colors which were given identical or highly similar names by a large number of survey participants, which would then serve as an approximate list of the most common colors rendered similarly across a range of computer monitors. Over 200,000 people eventually completed the survey,[26] and Munroe published the resulting list of 954 named RGB web colors[27] on the xkcd website. They have since been adopted as conventional color identifiers in various programming and markup languages, including Python and LaTeX.

/r9k/ is a board which implements Randall Munroe's "ROBOT9000" algorithm, where no exact reposts are permitted.[90][91] It is credited as the origin of the "greentext" rhetorical style which often center around stories of social interactions and resulting ineptness.

Several analyses have found a significantly decreased risk of coronary artery disease (CAD) in individuals with GS.[22][23]

Typically no treatment is needed.[1] If jaundice is significant phenobarbital may be used, which aids in the conjugation of bilirubin.

Gilbert syndrome (GS) is a syndrome in which the liver of affected individuals processes bilirubin more slowly than the majority.[1] It confers significant health benefits (see below). Many people never have symptoms.[1] Occasionally jaundice (a slight yellowish color of the skin or whites of the eyes) may occur.

The mild increase in unconjugated bilirubin due to Gilbert syndrome is closely related to the reduction in the prevalence of chronic diseases, especially cardiovascular disease and type 2 diabetes, related risk factors, and all-cause mortality.[20] Observational studies emphasize that the antioxidant effects of unconjugated bilirubin may bring survival benefits to patients.

Following a Mediterranean diet can reduce liver fat even without weight loss and is the most recommended dietary pattern for NAFLD. The Mediterranean diet is characterised by reduced carbohydrate intake, especially sugars and refined carbohydrates (40% of the calories vs. 50–60% in a typical low fat diet), and increased monounsaturated and omega-3 fatty acid intake (40% of the calories as fat vs. up-to 30% in a typical low fat diet). Both TV sitting (a reliable marker of overall sedentary behaviour) and physical activity are associated with cardio-metabolic health, NAFLD and overall mortality. A 'triple hit behavioural phenotype' of: i) sedentary behaviour, ii) low physical activity, and iii) poor diet have been defined. Clinical evidence strongly supports the role of lifestyle modification as a primary therapy for the management of NAFLD and NASH. This should be accompanied by the implementation of strategies to avoid relapse and weight regain.

Lifestyle intervention can be effective when treating non-alcoholic fatty liver diseases (NAFLD) patients. Weight loss decreases cardiovascular and diabetes risk and can also regress liver disease. Weight reductions of ⩾10% can induce a near universal non-alcoholic steatohepatitis resolution and fibrosis improvement by at least one stage. However, modest weight loss (>5%) can also produce important benefits on the components of the NAFLD activity score (NAS).

* no take away, delivery, fast food, or prepared foods * no soda or caloric drinks (regular snapple, juices, coffee drinks, etc can pack tons of sugar and calories yet zero nutrition) * veggies at every meal, and lots of them

Fatty liver is easy to reverse, mostly through diet. Eat more vegetables, fruits and fiber (lentils, nuts), add oatmeal to your diet. Limit processed foods especially things that have long shelf life as they are full of trans fat. You can also try intermittent fasting. Good luck!

In conclusion, NAFLD should not be neglected in normal weight subjects with altered liver enzymes. It could indicate a higher risk for metabolic disturbances and/or cardiovascular morbidity, or it could unravel a different entity of non-metabolic, non-alcoholic fatty liver disease. Accordingly, a deeper diagnostic investigation for secondary causes of hepatic steatosis, like hypobetalipoproteinemia and malabsorption diseases is required in lean NAFLD patients. Follow-up studies specifically in this set of patients is necessary in order to better understand the association with other diseases and the real prognosis of liver disease. Also, a difficult unsolved issue is how to treat these patients who already have normal weight and whether diet and exercise are useful.

Nonalcoholic fatty liver disease (NAFLD) has widely been considered a manifestation of metabolic syndrome in close relation to obesity [1]. In fact, although obesity is undoubtedly one of the main risk factors for fatty liver, since its first description it is known that it can also occur in lean subjects.

Conclusions : Lean-NAFLD is a new unrecognized clinicopathological entity, a frequent cause of cryptogenic liver disease

Nonalcoholic fatty liver disease (NAFLD) is commonly diagnosed in obese or overweight individuals. However, lean individuals with NAFLD are not rare but represent one significant end of the phenotypic spectrum of NAFLD. Although initial observations between obese and lean NAFLD reveal some metabolic parallels, these associations vary widely given differences in study populations and metabolic parameters assessed. The role of body composition in risk assessment is significant and incompletely assessed during most clinical encounters.

Despite these limitations, the study by Hagström et al. provides important insights into our understanding of the natural history of lean NAFLD. Even though patients with NAFLD who are lean have no increased risk of mortality (hazard ratio, 1.04), the rates of progression to severe liver disease (most notably cirrhosis) highlight that lean NAFLD is not a simple benign condition.

Nonalcoholic fatty liver disease (NAFLD) is a worldwide epidemic with global prevalence estimated at 25%.1 Although NAFLD is usually associated with obesity, patients who are not obese can also present with NAFLD; this is known as "lean NAFLD." Lean NAFLD is defined as NAFLD that develops in patients with a body mass index (BMI) <25 kg/m2. The prevalence of lean NAFLD varies from 7% in the United States2 to as high as 19% in Asia.3, 4 Unfortunately, being lean (not overweight) does not necessarily lead to better health.

I was a "lean" fatty liver. I have reversed it by cutting out alcohol, avoiding all processed foods, no sugar, no white flour, very low carb. Only fruit I ate was berries. Also, full fat products. No low fat or no fat. I also did intermittent fasting 16:8. It took 11 months.